SOCIETY MEETINGS. 
599 
disease which affects our domestic animals. Here we observe 
the characteristic fever, inflammation of digestive and respiratory 
organs, haemorrhagic lesions, and affected secretory surfaces. 
Severe indeed must be the suffering of a “ cholera ” hog. 
One who has lived through an attack of typhoid fever can¬ 
not for a long time afterwards notice a hog with well-developed 
cholera without a returning realization of the suffering then en¬ 
dured. The poor hog plainly gives evidence of suffering from 
the same headache, the same dull, aching, watery, sticky eyes, 
the same intense fever, dysentery, painful micturation, with 
their accompanying anorexia and disinclination to move. And 
why should he not? The bacillus of hog-cholera and the bacil¬ 
lus tyhi abdominalis are very like in every way. They grow 
readily in the laboratory upon the common culture media, mak¬ 
ing a most luxuriant growth upon potato. They may be prop¬ 
agated with equal facility in milk, and it matters not whether 
this milk is in the incubator of a laboratory or in a crock on the 
pantry-shelf. The colonies present macroscopically a dense cen¬ 
tre with attenuated borders. There is nothing very character¬ 
istic or interesting in this, but just examine some well-prepared 
sections from this colony under a good microscope, and how 
very quickly the whole scene changes. Of all the crazy scurry¬ 
ing crowds, the one now brought to view takes the cake. They 
are described in the text-books as actively motile, and they 
thoroughly warrant such description. Each one hustles across 
the field in tumultuous haste, as though he believed a “ bad 
black man ” was right after him. No wonder that these organ¬ 
isms permeate the entire body of their victim, or that death so 
often steps in and ends their journeying. 
Hog-cholera is one of the true septicsemic diseases. The 
organisms enter the general circulation and are conveyed to all 
vascular parts of the body. This advantage once gained, they 
multiply rapidly and cling to the vessel-walls wherever they are 
able to do so. They often gather in such numbers as to form 
plugs in the blood capillaries which result in haemorrhages. 
They lodge in the lympli-glands, in the capillary plexuses of the 
lungs, along the intestinal walls, and wherever lodged they pro¬ 
duce their toxic excretions. 
This oro-anism offers extreme resistance to the effects of 
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cold. Freezing does not seem to destroy it. Hence, we must 
conclude, that it is unsafe to depend on freezing wintry 
weather to stop an outbreak of cholera. 
The theory has been advanced by Gaffky that these typhoid 
