42 
J. MYERS. 
acunoe infiltrated with a bloody serum which when the integument is 
removed (the periosteum is readily detached at some places and at 
others firmly adherent) percolates and evaporates, causing the same 
bone to dry up within two or three months, so that it loses two thirds of 
its former weight without reducing its volume any. A diseased bone of 
this kind can be cut without any difficulty similar to cartilage, is elastic 
and tender in wet condition, when dried, fragile and proned to 
fracture. The muscles are relaxed, pale and intersected with consid¬ 
erable amount of adipous tissue. Lungs and heart are not markedly 
affected neither the spleen and intestines. There are ecchymosed patches 
in the mesentery. The liver which at the onset is hypersemic (found 
it in one case atrophied, of an ash gray color and tough, the hepatic 
vessels and ducts shrunken) becomes frail and even partly decayed. 
The kidneys at the beginning are hypersemic, later anaemic and 
relaxed, the pelvis often contained purulent mucous; it has also occur¬ 
red that partial decomposition had taken place. The bladder gener¬ 
ally contains an opaque vicid urine, mucous lining slackened and lead 
colored. I usually found that where there was a rupture of flexor 
tendons and ligaments, fragments of the coffin and navicular bones 
were attached; the excavations made by the disunited fragments of 
the above named bones, bore traces of a vehement congestion which 
must have taken place. 
In consequence of the unlimited duration of the disease which 
governs the different pathological lesions, I am unable to do justice in 
describing the intercurrent complications. What appeared remarkable 
to me. was the more solid consistency of the blood which may probably 
be attributed to the diminished desire for water that the animal had. 
Analysis of the same might possibly be of diagnostic value. 
The course of this disease is of a chronic nature and may linger 
from 2 to 3 months or more, lest hectic fever intervenes which brings 
on a speedy termination, or that lumbago or rupture of the flexor 
tendons demands destruction. The convalescence is seldom complete, 
the structural changes of the maxillary bones are permanent and in 
cases where they are not involved the disease is merely characterized 
by lameness of the extremities and impaired or changeable appetite; 
a re-occurrence may be anticipated. 
At the onset, the diagnosis is very obscure (as is the case in a num¬ 
ber of other diseases), until the force of the disease is exploded by a 
regular rheumatic attack. The variety of forms in which this enemy 
