190 
E. SEMMER. 
with air impregnated with foul elements, and the infusion took place one 
hour after the blood was taken from the cow. One of them died three 
days, the other five after the infusion took place. Both manifested 
septicaemia in high degree. The same blood which was injected into 
the other animals in the pure air, and caused no disadvantageous con¬ 
ditions, by these, where the operation took place in polluted air, caused 
septicaemia and a rapid death. Two rabbits were subcutaneously in¬ 
jected witji blood from a horse which had been inoculated with gland¬ 
ers four days previously. The blood was previously to injection brought 
through a room the air of which was polluted with foul elements. Both 
died after eighteen and twenty-four hours, and presented the phenom¬ 
ena of fully developed septic and putrid blood decomposition. The 
blood of the rabbits examined immediately after death contained 
scarcely any red cells, only detritus masses of the same, and a great 
many micrococcen and streptococcen, which must also have been pre¬ 
sent intra vitam, so many being present when the obduction was made 
which immediately followed their death. 
If the examination of the blood of animals suffering from putrid or 
septic decomposition of the same, does not always show bacteria intra 
vitam, the same does not prove that they are absolutely absent. The 
same may be disturbed by means of the active oxydination taking place 
in the circulating blood. It is easily supposable that the same may be 
filtered from the blood by means of the liver, kidneys and spleen, and 
yet they may increase, and periodically appear in the circulation in 
great numbers, until they finally gain the upper hand, and are able to so 
carry on the work of disintegrating the red cells. The circumstances 
that foulness bacteria very often develop in the cadaver very rapidly, 
and cause the disappearance of those peculiar to certain diseases has 
often led to many false conclusions. It is entirely wrong, as may be 
asserted, that there is no difference between the bacteria met with by 
different contagious diseases, and that the same are exclusively the con¬ 
sequence of the disease—only gradually varying according to the disease, 
and species of animals. 
As examples of the specific forms of bacteria by single diseases, I 
quote cattle. By the same we can distinguish—putrid bacteria, Fig. 1 
and 2; septic bacteria, Fig. 3 and 4 ; rabies bacteria, Fig. 4; anthrax 
oacteria, big. 5; rinderpest bacteria, Fig. 9. The bacteria by the rin¬ 
derpest were constituted by me in 1871. (Wiener Vierstelfahrschrift, 
1871, Bd. 36, S. 176), then from Klebs and Hallier, and again more ex- 
actingly treated by me in 1875. (Ueher die pathologische anatomie de 
Rinderpest Dorpat, 1875.) 
