PATHOLOGY OF PULMONARY DISEASES OF THE HORSE. 
69 
not suffice to develop an inflammatory process in the lungs, but it is 
sufficient when the predisposition is present, which we have been 
considering as due to emaciation, etc. I cannot look upon 
gelatinous infiltration in such cases, as the expression of a deutero- 
pathic pneumonia; as by horses diseased with malleus, it stands in 
no direct relation with that disease. Pneumonia develops entirely 
independent of the latter, and by the side of the same, and in 
animals by which malleus is not present. Although scrophulous 
individuals become easily sick and are frequently subject to pneu¬ 
monia, yet no pathologist would be inclined to designate such a 
pneumonia as deuteropathic; it is just as protopathic as every 
other pneumonia which develops by healthy individuals after 
violent irritation. Malleus predisposes the pulmonary tissues to 
gelatinous infiltration, because it causes the development of 
atelectasis and ischaemia, and it generates further a predisposi¬ 
tion to inflammatory processes in the lungs. The last are, how¬ 
ever, conditioned by simple actiological momenta, i. e ., such 
which in general cet. par. would be likely to produce inflamma¬ 
tory processes in the lungs. Pneumonia or gelatinous infiltra¬ 
tion is here only a complication with malleus, and must not be 
classed with the special processes of the latter. 
A second form of gelatinous infiltration stands, however, in 
intimate relation to the processes of malleus in the lungs. I can¬ 
not, however, in this place go into a minute discussion of malleus, 
but I will remark, that in every place, where the ulcerative pro¬ 
cesses of malleus exist, in most cases we are enabled to demon¬ 
strate the presence at the same time of simple inflammatory pro¬ 
cesses. For instance, we have upon the mucosa of the cavitates 
nasi, at the same time with the chancre (ulcus) of malleus, a simple 
catarrh upon that part of the mucosa complicated by the malleosic 
processes; in all places we may observe this combination of specific 
and simple inflammatory processes. The catarrhalic inflamed parts 
of the mucosa give the masses for expectoration. I will not deny 
that the ulcers ( chancres ) do not also give a secrete , but their secre¬ 
tion is but insignificant; the greater part of the secretion is given 
by the simple catarrhalic diseased parts of the mucosa. When the 
chancres (ulcers) of malleus penetrate deeply into the underlying 
