70 
F. S* BILLINGS. 
tissues along the larger part (in point of diameter) of the respira¬ 
tory tractus, they produce perichondritis , and upon the smaller 
parts peribronchitis or a broncho-pnemonia. These profusely 
situated processes have nothing specific about them, being purely 
inflammatory in character. We may observe by the presence of 
malleosic processes in the bronchioli resp. the termination 
of the same, a simple inflammatory process in the neighboring 
alveolae. This combination of specific and non-specific processes 
was first made clear by Yicliow, and later also by Boloff and 
Bollinger. The malleosic neoplasmata are to be mostly found in 
the parietes of the bronchi, and, indeed, as Gerlach was the first 
to show, in the ends of the bronchioli, and by the exceeding 
delicacy of the parietes of the bronchioli, it is easy for the 
processes of irritation to extend themselves to the tissues in the 
vicinity. The adjoining alveolae are, however, atelectatic, owing 
to the early obstruction of the lumina of the bronchi by the 
malleosic processes in the parietes of the same, and further to 
the muco-purulent secrete of the bronchial mucosa. The 
development of atelectasis may be accelerated by the concomi¬ 
tant presence of insufficient respiratory movements, as well as 
emaciated cachectic animals. The adjoining alveolae are also 
ischaemic. This ischaemia is the consequence of the hyperplasia 
of the connective tissue of the adventitia of the vessels (capil¬ 
lary ramifications of the a. pulmonalis). We can demonstrate this 
hyperplasia regularly by malleus in the peri-broncliial and peri¬ 
vascular connective tissue, and the adventia of the vessels in 
question is especially strongly complicated. By the hyperplasia 
of the connective tissue (admentitia) the vessels become com¬ 
pressed, aud those parts of the lungs ischaemia which derive their 
blood from the vessels in question. This paleness is especially 
marked when the animals in question are at the same time 
emaciated and ischaemic. For the same reasons the peri-bron¬ 
chial alveolae are atelectatic and ischaemie by horses complicated 
with malleosic bronchitis and peri-bronchitis. We may always 
be able to demonstrate around malleosic diseased bronchi, a circle 
of gelatinous infiltrated pulmonary tissue. This gelatinous in¬ 
filtration is of a deuteropathic origin, and as it is of malleosic 
