PATHOLOGY OF PULMONARY DISEASES OF THE HORSE. 
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origin, it may be designated as malleosic gelatinous infiltration 
(or gelatinous infiltration of glanders). The malleosic processes 
owe their genesis to a specific contagion, and it is therefore 
solely dependent on the intensity of the irritation, if the products 
are of a specific or simple inflammatory nature. Both forms of 
development are to be found in the course of malleus, and the 
malleosic gelatinous infiltration correspond to the simple slight 
conditions of irritation. 
There is also a proto- and a deuteropathic gelatinous infil¬ 
tration. Both represent inflammatory processes in atelectatic and 
ischaemic pulmonary tissue. I explain the genesis of the first by 
the complication of malleus with an acute pneumonia; and the 
last by the extension of the malleosic processes to the surround¬ 
ings. As, however, the malleosic bronchial complications are 
generally multiplex present, and as in general many bronchi 
lying in close vicinity to each other are complicated concomi¬ 
tantly, so must the peri and inter-bronchial pulmonary tissue 
always show a state of gelatinous infiltration. I must not forget 
to mention that I have frequently found a condition of full 
hepatization—complete cellular infiltration of the alveolae—in the 
alveolae in the immediate vicinity of the malleosic diseased 
bronchi, as an indication of intensive irritation, and that the 
gelatinous infiltration was first apparent outside of these changes 
at a considerable distance from the bronchi. These processes 
must be classed with those of broncho-pneumonia, the aetiology 
of which cannot easily escape us if we take into consideration 
to concomitantly existing malleosic bronchial complications. 
Primary gelatinous infiltration is a more extended process, and 
develops independent of any existing malleosic complications of 
the pulmonary tissues. The connection with the changes of 
malleus fails by protopathic gelatinous infiltration, and it is the 
presence or absence of this connection which I must demand 
respect in fixing a deferential necroscopical diagnosis ; for the 
primary inflammatory processes may (as the history of pneumonia 
teaches), present a lobular extension. With regard to the 
termination of gelatinous infiltration, only those forms can here 
interest us which have to do with the primary form of these 
