-90 
SPOLIA ZEYLANICA. 
When the monozoite has issued in the usual way from the 
cytocyst the latter remains within the corpuscle, sometimes 
showing a slight crumpling due to collapse of the membrane. 
The corpuscle becomes enlarged about twice the normal size, and 
its cytoplasm appears pale as if undergoing dissolution, which 
indeed is obviously the case. 
We have not been able to follow the stages in the involution of 
the cytocyst, but we do not doubt that it eventually disintegrates 
as its term of service has clearly expired. In one case only have 
we found a corpuscle which appears to contain the ghost of a 
cytocyst. 
With regard to the red-stained granules of the cytocyst, we 
regard these as belonging to a thin layer of residual protoplasm 
which is left round the periphery of the mother-cell after the 
formation of the axial monozoite. Sometimes the portion of the 
issuing parasite which has already emerged from the cytocyst 
appears reddish outside, the portion inside the sheath being pale 
blue. This would seem to be due to a slight amount of the 
residual protoplasm being carried out by the monozoite; or 
perhaps the cytoplasm of the corpuscle may be involved as well. 
Conclusion. 
We shall not attempt to discuss in detail the nature of the 
various forms we have described, believing that the observations 
in themselves are sufficiently striking to warrant publication. 
Of course there is the question of the genetic connection 
between the trophozoites of the first or endoglobular infection 
and the cytocysts and monozoites of the free infection. The 
evidence that they belong to the cycle of development of one and 
the same species is only presumptive. 
First of all there are the facts of the specific identity of the 
hosts, of the close proximity (within the radius of a few miles) 
of the localities, the fact that the parasites belong to the family 
Hsemogregarinidae, and lastly the circumstance of the approxi¬ 
mately synchronous development of the parasites in their respec¬ 
tive hosts, the trophozoites of the endoglobular infection present* 
ing the same phase of growth within narrow limits and the 
cytocysts of the free infection varying only within the limits of 
the origin and birth of the monozoites. 
As for the motile monozoites, their fate remains for the present 
a mystery and we will only hazard the suggestion that they 
represent fully developed forms ripe for transmission to another 
host. 
