in males from eggs dipped in 5 mg. D.E.S./100 ml. of o11. Also, 
eet Yee delay in spermatogenesis as compared to controls 
adle e 
Table II*# 
Effect of Hormone Treatment of the Eggs on Reproductive 
Organs of Genetic Males, (Males 6 months of age ) 
Treatment Body wt. (gm. ) Gonad wt. (em.) 
Control 3,112 17.07 | 
O11 emulsion 3,503 21.16 
D.E.S. 5mg./100ml. 3,545 9.19% 
oil emulsion 
¥significant at the 0.01 Level 
##van Teinhoven (1957) 
Herrick and Adams (1956) found that progesterone and/or 
diethylstilbestrol depressed testes weight (Table III). 
Table III## 
Testes Weight in Grams and Spermatogensis of S.C. White. 
Leghorn Males Treated with D.E.S. and/or progesterone 
Control* Progesterone Dabede De & P. 



‘wt. sperm Wte sperm wt. sperm wt. sperm 
20.8 + 10.6 R 1.4 - Oe? - 
* 21 birds used in each treatment 
*# Herrick and Adams (1956) 
+ normal R reduced - lacking 
The administration of progesterone or prolactin is antagonistic 
to gametogenesis, and also inhibits gonadotropic hormone 
secretion (Bates et al. (1937). Fox (1955) also noted that 
progesterone reduced comb index and testis weight of S.C. White 
Leghorns when progesterone was injected weekly. In the absence 
of the thyroid gland, Brown Leghorn testis development was sub- 
normal and spermatogenesis did not proceed beyond the formation 
of secondary spermatocytes (Blivaiss, 1947). Although Mellen 
and Wentworth (1962) noted some mating activity of radio- 
thyroidectomized males, they did not present any data pertaining 
to spermatogenesis. Adrenaline appears to have an unfavorable 
influence on the gonads. Its injection has been found to affect 
the production of spermatozoa very adversely in domestic cocks 
(Wheeler et al., 1942) and in House Sparrows (Perry, 1941). 
Cortisone induced /precocious spermatogenesis in cockerels and 
Pekin ducks (Leroy, 1952; cited by Romanoff, 1960). 
78 
