

in 

DOMINANCE. REVERSED DOMINANCE RECESSIVENESS, ETC. (225) 33 
For a mendelian explanation of table VI we may consider the par- 
ents as hereditary variations (homozygotes) and the children as hete- 
rozygotes which are more or less intermediate, so according to the 
formula DD x RR = DR.» 
When we recollect that we already had the opportunity to remark 
that there are also extreme indices which in connection with the in- 
dices of parents cannot be homozygous (p. 25), then for table VI, 
too, we must expect that among the parents there will also be hetero- 
zygotes. This is obvious, when taking in view those families of which 
one or more of the grand-parents are known. One of the parents is a 
heterozygote in the families 73c, 304, 335c, 301b, 240c, 73b, 285 and 
257. Both parents can be homozygotes in the families 34b, 271a, 20, 
2212851. 296,334, 308, 232b, 233,..16b, 64, 87e, 147, 324e, 331b, 
and 149. 
Further we must consider that for a mendelian interpretation the 
presence of a reccessive microbrachycephalic index (according to the 
result of table V) can still have some importance. 
Of the different families of table VI, family 32g with small mutual 
differences of the indices of parents, of 3 grand-parents and of 10 
children might also be classified into table IV. 
When of the above mentioned group of 8 families for which we might 
accept segregation according to the formula RR x DR = RR + DR 
and which consequently might also be taken up into the tables IT and 
III, adding the children which show the same characteristics, we can 
find the 39 children divided into 2 groups of 19 and 20 children. This 
addition is however in some degree arbitrary (see p. 9). 
Table VII. The indices of parents differ much; those of children 
follow the parent with the high index, 43 cases and still 7 cases of in 
complete families. (p. 122). 
In these cases the high index is dominant over the low one or in 
general brachycephaly is dominant over dolichocepahly (see also 
BRYN, 1920, p. 202). 
For the spreading of children in each family we refer to the curves 
of figure 7a, p. 192. Here are also hereditary variations (fig. Jor 
Table VIII. There is a large difference between the indices 
of parents; the children follow the parent with the low index, 21 
cases and still 7 of incomplete families. See the curves of fig. 8a (p. 193) 
and fig. 8. 
(15) 3 
